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A the latest discovery highlighted a novel position for intracellular MIF in regulating the accumulation of misfolded Cu/Zn superoxide dismutase (SOD1). Mutations in SOD1 are connected with twenty% of your cases of familial amyotrophic lateral sclerosis, which can be characterized by lack of motor neurons [seventy two]. Though MIF was found as a f
Two polymorphisms from the human MIF gene have already been associated with human health conditions. a single is only one-nucleotide mutation (a G-to-C changeover at place −173) inside the five′ flanking location, that's connected to systemic-onset juvenile arthritis15. another polymorphism is a CATT-tetranucleotide repeat at place −794, whic